Five years ago we reported on what we thought was an important clue in the search for understanding the well-documented association between childhood leukemia and EMF exposure. A team based in Shanghai presented evidence that children carrying a genetic variation linked to DNA repair were four times more likely to develop leukemia than those without that genetic marker. We called the finding a “major breakthrough” and predicted, “It simply cannot be ignored.”
We were wrong. So wrong.
What happened next —or rather, what did not happen— sheds light on why EMF research treads water and never moves forward.
